Absorption of VD not only promotes the absorption of calcium and phosphorus in the intestine but also acts on bone tissue. Although wolfberry is not conducive to the absorption of VD due to its physiological characteristics, in the conventional feeding process, fish farmers often use fish as feed, and it is generally not easy to have a deficiency of VD. Early weaning diarrhea is due to calcium in the diet during artificial feeding. When the ratio of phosphorus and phosphorus is unregulated, when inorganic salts, vitamins and proteins are lacking, or when a person suffers from gastroenteritis, it is easy for the disease to occur in the dark and unclean cages due to lack of ultraviolet radiation. The clinical manifestations of VD deficiency are mainly digestive disorders, lameness, dorsiflexion, vertebrae and facial bones, and enlarged limb joints, which can lead to death. According to the treatment of 8 cases of 貉VD deficiency treated by the clinic, the author summarized the following:
1 Pathogenesis
VD is a steroidal substance with many types, of which the main ones are VD2 (ergocalciferol) and VD3 (cholecalciferol). VD2 is a product that is irradiated by ultraviolet rays from yeast or ergosterol in ergot; VD3 is a product that is irradiated with ultraviolet light from 7-deoxycholesterol in animal skin and fat. After VD is absorbed by the body, it can only exert its physiological function after being activated by hydroxylation. Animals do not suffer from VD deficiency under normal living conditions and feeding conditions. Therefore, the lack of ultraviolet radiation, the lack of calcium in food, and the inappropriate proportion of calcium and phosphorus are the main factors that cause sputum VD deficiency.
2 clinical symptoms
In the early stages of suffering from cramps, appetite loss, poorer spirit, indigestion, tangled hairs, weakness in the hind limbs, and lack of exercise are generally shown. About 4 weeks later, the anterior and posterior limbs of the affected triceratops couldn't be normal weight-bearing and crawled. The long bones of the vertebrae and limbs were deformed, the arches back, and the joints became bigger. At the same time, there were serious digestive dysfunctions. Constipation or constipation alternated with diarrhea, but constipation was more common. . Severe late failure died.
3 treatment measures
Clinically, according to the medical history and clinical symptoms, the conditional combination of X-ray examination results can be basically confirmed.
3.1 intramuscular injection of VD 2 million IU / time, once a week for 3 to 5 times. Oral ossification of triol can also be supplemented with calcium.
3.2 In case of severe disease or poor efficacy, nandrolone, anabolic hormone phenylpropionate, is injected intramuscularly every time for 2 to 4 mL every 2 weeks. At the same time, the protein content in the feed is appropriately increased so that it can exert its assimilation.
3.3 Intramuscular injection of Vidin calcium, 2mL each time, 1 day, once every 5 to 7 times. At the same time, moderate amounts of bone meal are added to the diet. After one month of treatment, the condition improved markedly, but individuals with heavier disease still showed varying degrees of lameness.
4 Summary
4.1 The efficacy of this disease is related to the length of the disease and the severity of the disease. The shorter the course of disease, the lighter the disease, the more timely the treatment, the better the effect; on the contrary, the efficacy is poor.
4.2 VD can not only promote the absorption of calcium and phosphorus in the intestine, but also act on the bone tissue. The lack of VD in the growing pups can induce rickets; therefore, VD should also be considered for the treatment of 貉VD deficiency. .
4.3 Scientific feeding and prevention of partial eclipse are important measures to prevent this disease.
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