U.S. researchers have recently performed genetic engineering experiments on experimental mice, successfully providing them with the ability to resist the pathogens of spongiform encephalopathy. This discovery was seen by some scientists as a "breakthrough" in the field of mad cow disease research. Mad cow disease and its manifestations in the human body, such as the new Creutzfeldt-Jakob disease and other spongiform encephalopathy, are caused by mutations in sawn protein (also known as the Prion protein). In mammals, including humans, there is serin in the body. It is harmless under normal conditions, but after a specific mutation occurs, it can lead to dementia-type transmissible spongiform encephalopathy in humans or animals. Earlier studies have shown that mutations in normal sawn proteins are not all pathogenic, and that certain variants of the protein can be "prevented" and protect animals from pathogenic sawing proteins. Dr. Safar, from the University of California, San Francisco, used mice as a model to provide further evidence. Safar and others reported in a new issue of the American Journal of the National Academy of Sciences that they first conducted genetic engineering experiments on mice in the study, resulting in significant changes in the amino acids that constitute normal sawn proteins in their bodies. After these mice matured, the researchers injected high doses of the diseased sawin and found that although some of them produced pathogenic serin in the body, none of the mice showed a complete spongy appearance. Encephalopathy symptoms. At the same time, the researchers also selected a group of mice that carried normal serin in their bodies as control subjects. However, these mice developed lethal spongiform encephalopathy after receiving the injection of the diseased sawn protein. Some experts commented that the latest study by Safar and others showed that if spongiform encephalopathy cannot be eradicated, it is at least possible to be completely prevented. Safar said that their next step is to use similar genetic engineering methods to breed cattle that are resistant to mad cow disease.
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